Medical examination with ayurvedic diagnosis of constitution
examination with ayurvedic diagnosis of constitution,
making out of the therapy plan:
one-off sum: THB 1.000,00
Lectures for diet and daily routine
The art of diagnosis in
Ayurveda can be divided
into 2 parts: The inspection of the human being (Rogi-Pariksa) and the
examination of the disease (Roga-Pariksa). A very important aspect is the
determination of the constitution, which maybe described as the individual
state of health. The constitution is diagnosed by determining the
individual constellation of the 3 doshas at the time of birth. On the
basis of the dietary recommendations are given, and it also indicates the
proneness to possible diseases. A disease start by a deviation from the
constitution and is understood to represent a deviation from the normal
composition of the 3. Thus the current state of the 3
doshas has a great
importance in finding a diagnosis.
|Medical examination with ayurvedic diagnosis of constitution
|Medical examination with ayurvedic diagnosis of constitution
the application form here
Information about diagnostic methodology in
plenty but is lying scattered in the text at various places in the form of
Sutras, Slokas or Verses. This puts Ayurvedic scholars in difficulty for
examining and diagnosing a patient in an organized or systematic way. Many
new diseases are developing and so never diagnostic methodologies. It has
become essential that an alternative medicine should remain updated with
the recent developments in modern science utilized for diagnosing
diseases. According to Charaka, three things are essential for diagnosing
3.Aptopadesh (saying of seers in authoritative
classical and other texts)
Susruta prescribed, the use of five sense while examining a patient and
asking Prashnas (questioning). It is essential for a physician to do both
Roga (disease) and Rogi (patient) Pariksha (examination).
Madhava mentioned Nidanapanchak (five diagnostic aspects) which are:
Nidana or Hetu (causes responsible for Roga, the disease)
3. Rupa (Signs and symptoms of Roga)
Samprapti (Pathogenesis) and
5. Upasaya (Therapeutic test)
Madhava we find Astavidh Pariksha developed as in the text Yogaratnakara.
1. Nadi (pulse)
2. Mutra (urine)
4. Jihva (tongue)
5. Shabda (voice)
6. Sparsha (skin or
7. Drika (sight or eyes) and
8. Akriti (appearances, face,
In addition to the above in females, examination of Artava
(menstruation) and Stanya (breast milk) is done. Ayurveda texts mentioned Dashavidha Pariksha (ten types of examination). This examination should be
made before final medicaments are given to the patient. In this way we
observe, there is not much difference in the Ayurvedic methodology of
diagnosing from the modern allopathic method of diagnosing disease. Some
minor conceptual variations are there.
Cancer's Sweet Tooth
by Patrick Quillin,
PHD, RD, CNS
From The April 2000 Issue of Nutrition Science News
During the last 10 years I have worked with more than 500 cancer
patients as director of nutrition for Cancer Treatment Centers of
America in Tulsa, Okla. It puzzles me why the simple concept "sugar
feeds cancer" can be so dramatically overlooked as part of a
comprehensive cancer treatment plan.
Of the 4 million cancer patients being treated in America today, hardly
any are offered any scientifically guided nutrition therapy beyond being
told to "just eat good foods." Most patients I work with arrive with a
complete lack of nutritional advice. I believe many cancer patients
would have a major improvement in their outcome if they controlled the
supply of cancer's preferred fuel, glucose. By slowing the cancer's
growth, patients allow their immune systems and medical debulking
therapies -- chemotherapy, radiation and surgery to reduce the bulk of
the tumor mass -- to catch up to the disease. Controlling one's
blood-glucose levels through diet, supplements, exercise, meditation and
prescription drugs when necessary can be one of the most crucial
components to a cancer recovery program. The sound bite -- sugar feeds
cancer -- is simple. The explanation is a little more complex.
The 1931 Nobel laureate in medicine, German Otto Warburg, Ph.D., first
discovered that cancer cells have a fundamentally different energy
metabolism compared to healthy cells. The crux of his Nobel thesis was
that malignant tumors frequently exhibit an increase in anaerobic
glycolysis -- a process whereby glucose is used as a fuel by cancer
cells with lactic acid as an anaerobic byproduct -- compared to normal
tissues.1 The large amount of lactic acid produced by this fermentation
of glucose from cancer cells is then transported to the liver. This
conversion of glucose to lactate generates a lower, more acidic pH in
cancerous tissues as well as overall physical fatigue from lactic acid
buildup.2,3 Thus, larger tumors tend to exhibit a more acidic pH.4
This inefficient pathway for energy metabolism yields only 2 moles of
adenosine triphosphate (ATP) energy per mole of glucose, compared to 38
moles of ATP in the complete aerobic oxidation of glucose. By extracting
only about 5 percent (2 vs. 38 moles of ATP) of the available energy in
the food supply and the body's calorie stores, the cancer is "wasting"
energy, and the patient becomes tired and undernourished. This vicious
cycle increases body wasting.5 It is one reason why 40 percent of cancer
patients die from malnutrition, or cachexia.6
Hence, cancer therapies should encompass regulating blood-glucose levels
via diet, supplements, non-oral solutions for cachectic patients who
lose their appetite, medication, exercise, gradual weight loss and
stress reduction. Professional guidance and patient self-discipline are
crucial at this point in the cancer process. The quest is not to
eliminate sugars or carbohydrates from the diet but rather to control
blood glucose within a narrow range to help starve the cancer and
bolster immune function.
The glycemic index is a measure of how a given food affects
blood-glucose levels, with each food assigned a numbered rating. The
lower the rating, the slower the digestion and absorption process, which
provides a healthier, more gradual infusion of sugars into the
bloodstream. Conversely, a high rating means blood-glucose levels are
increased quickly, which stimulates the pancreas to secrete insulin to
drop blood-sugar levels. This rapid fluctuation of blood-sugar levels is
unhealthy because of the stress it places on the body (see glycemic
index chart, p. 166).
Sugar in the Body and Diet
Sugar is a generic term used to identify simple carbohydrates, which
includes monosaccharides such as fructose, glucose and galactose; and
disaccharides such as maltose and sucrose (white table sugar). Think of
these sugars as different-shaped bricks in a wall. When fructose is the
primary monosaccharide brick in the wall, the glycemic index registers
as healthier, since this simple sugar is slowly absorbed in the gut,
then converted to glucose in the liver. This makes for "time-release
foods," which offer a more gradual rise and fall in blood-glucose
levels. If glucose is the primary monosaccharide brick in the wall, the
glycemic index will be higher and less healthy for the individual. As
the brick wall is torn apart in digestion, the glucose is pumped across
the intestinal wall directly into the bloodstream, rapidly raising
blood-glucose levels. In other words, there is a "window of efficacy"
for glucose in the blood: levels too low make one feel lethargic and can
create clinical hypoglycemia; levels too high start creating the
rippling effect of diabetic health problems.
The 1997 American Diabetes Association blood-glucose standards consider
126 mg glucose/dL blood or greater to be diabetic; 126 mg/dL is impaired
glucose tolerance and less than 110 mg/dL is considered normal.
Meanwhile, the Paleolithic diet of our ancestors, which consisted of
lean meats, vegetables and small amounts of whole grains, nuts, seeds
and fruits, is estimated to have generated blood glucose levels between
60 and 90 mg/dL.7 Obviously, today's high-sugar diets are having
unhealthy effects as far as blood-sugar is concerned. Excess blood
glucose may initiate yeast overgrowth, blood vessel deterioration, heart
disease and other health conditions.8
Understanding and using the glycemic index is an important aspect of
diet modification for cancer patients. However, there is also evidence
that sugars may feed cancer more efficiently than starches (comprised of
long chains of simple sugars), making the index slightly misleading. A
study of rats fed diets with equal calories from sugars and starches,
for example, found the animals on the high-sugar diet developed more
cases of breast cancer.9 The glycemic index is a useful tool in guiding
the cancer patient toward a healthier diet, but it is not infallible. By
using the glycemic index alone, one could be led to thinking a cup of
white sugar is healthier than a baked potato. This is because the
glycemic index rating of a sugary food may be lower than that of a
starchy food. To be safe, I recommend less fruit, more vegetables, and
little to no refined sugars in the diet of cancer patients.
What the Literature Says
mouse model of human breast cancer demonstrated that tumors are
sensitive to blood-glucose levels. Sixty-eight mice were injected with
an aggressive strain of breast cancer, then fed diets to induce either
high blood-sugar (hyperglycemia), normoglycemia or low blood-sugar
(hypoglycemia). There was a dose-dependent response in which the lower
the blood glucose, the greater the survival rate. After 70 days, 8 of 24
hyperglycemic mice survived compared to 16 of 24 normoglycemic and 19 of
20 hypoglycemic.10 This suggests that regulating sugar intake is key to
slowing breast tumor growth (see chart, p. 164).
In a human study, 10 healthy people were assessed for fasting
blood-glucose levels and the phagocytic index of neutrophils, which
measures immune-cell ability to envelop and destroy invaders such as
cancer. Eating 100 g carbohydrates from glucose, sucrose, honey and
orange juice all significantly decreased the capacity of neutrophils to
engulf bacteria. Starch did not have this effect.11
four-year study at the National Institute of Public Health and
Environmental Protection in the Netherlands compared 111 biliary tract
cancer patients with 480 controls. Cancer risk associated with the
intake of sugars, independent of other energy sources, more than doubled
for the cancer patients.12 Furthermore, an epidemiological study in 21
modern countries that keep track of morbidity and mortality (Europe,
North America, Japan and others) revealed that sugar intake is a strong
risk factor that contributes to higher breast cancer rates, particularly
in older women.13
Limiting sugar consumption may not be the only line of defense. In fact,
an interesting botanical extract from the avocado plant (Persea
americana) is showing promise as a new cancer adjunct. When a purified
avocado extract called mannoheptulose was added to a number of tumor
cell lines tested in vitro by researchers in the Department of
Biochemistry at Oxford University in Britain, they found it inhibited
tumor cell glucose uptake by 25 to 75 percent, and it inhibited the
enzyme glucokinase responsible for glycolysis. It also inhibited the
growth rate of the cultured tumor cell lines. The same researchers gave
lab animals a 1.7 mg/g body weight dose of mannoheptulose for five days;
it reduced tumors by 65 to 79 percent.14 Based on these studies, there
is good reason to believe that avocado extract could help cancer
patients by limiting glucose to the tumor cells.
Since cancer cells derive most of their energy from anaerobic glycolysis,
Joseph Gold, M.D., director of the Syracuse (N.Y.) Cancer Research
Institute and former U.S. Air Force research physician, surmised that a
chemical called hydrazine sulfate, used in rocket fuel, could inhibit
the excessive gluconeogenesis (making sugar from amino acids) that
occurs in cachectic cancer patients. Gold's work demonstrated hydrazine
sulfate's ability to slow and reverse cachexia in advanced cancer
patients. A placebo-controlled trial followed 101 cancer patients taking
either 6 mg hydrazine sulfate three times/day or placebo. After one
month, 83 percent of hydrazine sulfate patients increased their weight,
compared to 53 percent on placebo.15 A similar study by the same
principal researchers, partly funded by the National Cancer Institute in
Bethesda, Md., followed 65 patients. Those who took hydrazine sulfate
and were in good physical condition before the study began lived an
average of 17 weeks longer.16
In 1990, I called the major cancer hospitals in the country looking for
some information on the crucial role of total parenteral nutrition (TPN)
in cancer patients. Some 40 percent of cancer patients die from
cachexia.5 Yet many starving cancer patients are offered either no
nutritional support or the standard TPN solution developed for intensive
care units. The solution provides 70 percent of the calories going into
the bloodstream in the form of glucose. All too often, I believe, these
high-glucose solutions for cachectic cancer patients do not help as much
as would TPN solutions with lower levels of glucose and higher levels of
amino acids and lipids. These solutions would allow the patient to build
strength and would not feed the tumor.17
The medical establishment may be missing the connection between sugar
and its role in tumorigenesis. Consider the million-dollar positive
emission tomography device, or PET scan, regarded as one of the ultimate
cancer-detection tools. PET scans use radioactively labeled glucose to
detect sugar-hungry tumor cells. PET scans are used to plot the progress
of cancer patients and to assess whether present protocols are
In Europe, the "sugar feeds cancer" concept is so well accepted that
oncologists, or cancer doctors, use the Systemic Cancer Multistep
Therapy (SCMT) protocol. Conceived by Manfred von Ardenne in Germany in
1965, SCMT entails injecting patients with glucose to increase
blood-glucose concentrations. This lowers pH values in cancer tissues
via lactic acid formation. In turn, this intensifies the thermal
sensitivity of the malignant tumors and also induces rapid growth of the
cancer. Patients are then given whole-body hyperthermia (42 C core
temperature) to further stress the cancer cells, followed by
chemotherapy or radiation.19 SCMT was tested on 103 patients with
metastasized cancer or recurrent primary tumors in a clinical phase-I
study at the Von Ardenne Institute of Applied Medical Research in
Dresden, Germany. Five-year survival rates in SCMT-treated patients
increased by 25 to 50 percent, and the complete rate of tumor regression
increased by 30 to 50 percent.20 The protocol induces rapid growth of
the cancer, then treats the tumor with toxic therapies for a dramatic
improvement in outcome.
The irrefutable role of glucose in the growth and metastasis of cancer
cells can enhance many therapies. Some of these include diets designed
with the glycemic index in mind to regulate increases in blood glucose,
hence selectively starving the cancer cells; low-glucose TPN solutions;
avocado extract to inhibit glucose uptake in cancer cells; hydrazine
sulfate to inhibit gluconeogenesis in cancer cells; and SCMT.
female patient in her 50s, with lung cancer, came to our clinic, having
been given a death sentence by her Florida oncologist. She was
cooperative and understood the connection between nutrition and cancer.
She changed her diet considerably, leaving out 90 percent of the sugar
she used to eat. She found that wheat bread and oat cereal now had their
own wild sweetness, even without added sugar. With appropriately
restrained medical therapy -- including high-dose radiation targeted to
tumor sites and fractionated chemotherapy, a technique that distributes
the normal one large weekly chemo dose into a 60-hour infusion lasting
days -- a good attitude and an optimal nutrition program, she beat her
terminal lung cancer. I saw her the other day, five years later and
still disease-free, probably looking better than the doctor who told her
there was no hope.
Ph.D., R.D., C.N.S., is director of nutrition for Cancer Treatment
Centers of America in Tulsa, Okla., and author of Beating Cancer With
Nutrition (Nutrition Times Press, 1998).
Warburg O. On
the origin of cancer cells. Science 1956 Feb;123:309-14.
Volk T, et al. pH in human tumor xenografts: effect of intravenous administration
of glucose. Br J Cancer 1993 Sep;68(3):492-500.
Diet and cancer: markers, prevention and treatment. New York: Plenum
Press; 1994. p 203.
Leeper DB, et
al. Effect of i.v. glucose versus combined i.v. plus oral glucose on
human tumor extracellular pH for potential sensitization to thermoradiotherapy. Int J Hyperthermia 1998 May-Jun;14(3):257-69.
F, et al. Abnormal substrate metabolism and nutritional strategies
in cancer management. JPEN J Parenter Enteral Nutr 1991
Proper use and recognized role of TPN in the cancer patient.
Nutrition 1990 Jul-Aug;6(4 Suppl):6S-7S, 10S.
J, et al. The glucose revolution. Newport (RI) Marlowe and Co.;
et al. Glucotoxicity: potential mechanisms. Clin Geriatr Med 1999
Hoehn, SK, et
al. Complex versus simple carbohydrates and mammary tumors in mice.
Nutr Cancer 1979;1(3):27.
GA, et al. Glycemic modulation of tumor tolerance in a mouse model
of breast cancer. Biochem Biophys Res Commun 1985 Nov
Sanchez A, et
al. Role of sugars in human neutrophilic phagocytosis. Am J Clin
Nutr 1973 Nov;26(11):1180-4.
Moerman CJ, et
al. Dietary sugar intake in the aetiology of biliary tract cancer.
Int J Epidemiol 1993 Apr;22(2):207-14.
Seeley S. Diet
and breast cancer: the possible connection with sugar consumption.
Med Hypotheses 1983 Jul;11(3):319-27.
Board M, et
al. High Km glucose-phosphorylating (glucokinase) activities in a
range of tumor cell lines and inhibition of rates of tumor growth by
the specific enzyme inhibitor mannoheptulose. Cancer Res 1995 Aug
et al. Hydrazine sulfate in cancer patients with weight loss. A
placebo-controlled clinical experience. Cancer 1987 Feb
et al. Hydrazine sulfate influence on nutritional status and
survival in non-small-cell lung cancer. J Clin Oncol 1990
College of Physicians. Parenteral nutrition in patients receiving
cancer chemotherapy. Ann Intern Med 1989 May;110(9):734.
Potential role of FDG-PET imaging in understanding tumor-host
interaction. J Nucl Med 1995 May;36(5):893-9.
von Ardenne M.
Principles and concept 1993 of the Systemic Cancer Multistep Therapy
(SCMT). Extreme whole-body hyperthermia using the infrared-A
technique IRATHERM 2000 -- selective thermosensitisation by
hyperglycemia -- circulatory back-up by adapted hyperoxemia.
Strahlenther Onkol 1994 Oct;170(10):581-9.
et al. Evaluation of systemic tolerance of 42.0 degrees C infrared-A
whole-body hyperthermia in combination with hyperglycemia and
hyperoxemia. A Phase-I study. Strahlenther Onkol 1994
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